NEET Pg once again is in news. The Exam has been preponed.

This may alter the time strategy for many Neet PG Aspirants.

The Examination dates have been changed quite a bit. This adversely affects the students.

However  in view of the fact that the students can do nothing about it.

The students should not get too much disturbed.

The number of days for Certain subject preparation need to be reduced.

In such cases students have to be a bit more selective. There is a need for high yield content and best possible source of information about latest developments.

The students need to focus on all subjects, all important topics, Best Possible Explanations, High yield Points, Extra Edge and Image Based Text wherever applicable.

The Factors for NEET Pg students not doing well are:

• Not taking their Clinical postings seriously.
• Not Good sources of information.
• Relying on Advertisements and packages without consultation properly
• Studying from unnecessary video channels
• Not focusing on Standard Text materials
• Looking for short cuts to success

Reading from standard text books must be encouraged. Main topics from books like Harrisons medicine, Bailey surgery, Robbins Pathology, should be encouraged.

The Platinum Q Bank is a new addition to the Preparation for FMGE Students. New Students preparing for FMGE and NEET PG can benefit from the book as the book provides a Good q bank and the yield for exams can be high. The Questions are clinically oriented and some latest topics and image based questions have been added. The Book needs two to three revisions.

Examples of Questions from Platinum Q Bank (With Permission of Author)

Q: A 68 year old is examined by Neurologist. He has inability to recognize objects by feeling their shape and neglect of contralateral body parts. Most likely the lesion is in:

1. Midbrain
2. Parietal lobe lesions
3. Temporal lobe lesions
4. Prefrontal cortical lesions

Ans B Parietal lobe lesions

Parietal lobe lesions

• Parietal lobe lesions are characterized by a visual field attention deficit in the contralateral visual field; a left-sided parietal lobe lesion produces an inability to see movement of fingers in the patient’s tight visual field when fingers in both visual fields are moved.
• Patients exhibit contralateral astereognosis (inability to recognize objects by feeling their shape) and neglect of contralateral body parts.
• If the lesion is present on the dominant side, finger agnosia may be present (inability to name fingers).

Q: A young child is diagnosed with Mc Ardle’s disease. This disease is usually due to

1. Liver Phosphorylase deficiency
2. Muscle  Phosphorylase deficiency
3. Lysosomal alpha-1,4-glucosidase deficiency
4. G6PD Deficiency

Ans B Muscle   Phosphorylase deficiency

•  “Mc Ardle disease “This disorder is characterized by increasing intolerance for strenuous exercise.
• Strenuous muscle activity is accompanied by severe cramps and may be followed by myoglobinuria, which can precipitate anuria and renal failure. In middle life, the fatigue increases and muscle wasting and weakness predominate.
• The serum CPK may be permanently or intermittently elevated.
• Muscle exercise is normally accompanied by release of lactate and of inosine, hypoxanthine and ammonia through the purine nucleotide cycle. In myophosphorylase deficiency lactic acid production is blocked and release of the purine nucleotide cycle compounds is exaggerated. The ensuing myogenic hyperurecemia is one of the characteristic features of defects of muscle glycogenosis.
• Phosphorylase activity must be assayed in muscle;
• Liver Phosphorylase is presumably normal, as is glucose homeostasis.
• DNA mutation analysis is useful in myophosphorylase deficiency as there are common mutations.

Q: A 45 year old male is admitted in Medicine ward with Pericardial Effusion. His vital signs are: Temp:  98⁰F, BP: 90/70 mmHg, PR: 133/m and RR 16/min. CXR Demonstrates Effusion. Most likely he will present with:

1. hypertension, tachycardia, distended neck veins, indistinct heart sounds
2. hypotension, bradycardia, distended neck veins, indistinct heart sounds
3. hypotension, tachycardia, distended neck veins, distinct heart sounds
4. hypotension, tachycardia, distended neck veins, indistinct heart sounds

Ans D hypotension, tachycardia, distended neck veins, indistinct heart sounds

Pericardial effusion. Clinical features are:

• A small effusion produces no symptoms.
• Large effusions lead to cardiac tamponade (hypotension, tachycardia, distended neck veins, indistinct heart sounds).
• Heart sounds are diminished, and it is difficult to locate the point of maximal impulse.
• Friction rub secondary to coexistent pericarditis is heard.

Q: A 60 year old patient presented in an endocrine clinic with Addison’s disease and is in a state of altered consciousness. He is dehydrated and has hypotension and seems to be in crisis. The Best treatment would be by:

1. Fludrocortisone +Saline + Glucocorticoids
2. Thiamine + Hydrocortisone
3. Glucose IV + Thiamine + Predinosolone
4. Fludrocortisone +Glucose IV + Thiamine

Ans A Fludrocortisone +Saline + Glucocorticoids

Treatment   of Addison’s disease:

• Glucocorticoid replacement is required; increased dosages are required during stressful periods (e.g., surgery or illness).
• Mineralocorticoid replacement (Fludrocortisone,) is required in patients who have persistent Hyponatremia, hyperkalemia, and hypotension.
• Adrenal crisis is treated with 100 mg of cortisol infused over 5 to 10 minutes, followed by approximately 300 mg over the next 24 hours.
• Intravenous saline is also required to replenish lost electrolytes and volume.
• Mineralocorticoids may be necessary if hypotension and dehydration persist.

Q: A Diabetic patient presented with acute onset fever and dysuria. There were associated chills and rigors. Urine examination revealed plenty of leucocytes with glycosuria. A  CT Scan of Abdomen was taken. Most Likely Diagnosis is:

1. Acute pyelonephitis
2. Chronic Pyelonephritis
3. Renal abscess
4. Emphysematous Pyelonephritis

Ans D  Emphysematous Pyelonephritis

Diabetics are prone to widely destructive, emphysematous pyelonephritis and renal papillary necrosis, renal carbuncle or perinephric abscess, xanthogranulomatous pyelonephritis. Diabetic patient presenting with acute onset fever and dysuria with Urine examination revealed plenty of leucocytes with glycosuria and A  CT Scan of Abdomen showing gas in kidneys clearly defines the condition.

Q: A 36 -year-old woman presented to the gynecologist with complaints of profuse vaginal discharge. There was no discharge from the cervix on the speculum examination. The diagnosis of bacterial vaginosis was made based upon all of the following findings on microscopy except:

            A.        Abundance of gram variable coccobacilli

            B.        Decreased number of lactobacilli

            C.        Abundance of polymorphs

            D.        Presence of clue cells

Ans C Abundance of polymorphs

Bacterial Vaginosis: The wet-mount in Bacterial Vaginosis is usually characterized by: clue cells, an abundance of bacteria of various morphologies, the absence of homogeneous bacilli (lactobacilli), and an absence or paucity of inflammatory cells.

Microscopy in Bacterial Vaginosis shows:

• Clue cell in wet mount.
• Increased Number of Gardenella vaginalis
• Decreased Number of lactobacilli
• Leucocytes  usually absent

These Examples of Questions from Platinum Q give you a rough idea for how to get an idea about the Question types asked in Examinations.

Links for Purchasing Platinum Q Bank  are :

https://notionpress.com/read/platinum-plus-q-bank-volume-i

https://notionpress.com/read/platinum-plus-q-bank-volume-ii

 

We hope that this helps you in getting an idea. For Online Q Bank you can directly sub scribe to:

https://www.medexamsprep.com/category/package/neet-pg

Your Prepration should be ahead of Your Examiners thoughts. Meaning that you should be well prepared.
In These circumstances we recommend you to go through Q Banks with atleast two revisons each.

Best of Luck for your Preponed Exams.